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The presence of hyper-phosphorylated tau protein, one of the hallmarks of both Alzheimer's disease and ALS–PDC of Guam, in motor neurons in lumbar spinal cord clearly suggests that additional pathological processes associated with aluminum are occurring.
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Given the above, it is not surprising that aluminum has been widely proposed as a factor in neurodegenerative diseases and has been found in association with degenerating neurons in specific CNS regions
Aluminum injected animals show severe anterograde degeneration of cholinergic terminals in cortex and hippocampus ¾åµ¤ò¹Íθ¤¹¤ë¤È¡¢¥¢¥ë¥ß¥Ë¥¦¥à¤¬¿À·ÐÊÑÀ¼À´µ¤ÎÍ×°ø¤È¤·¤Æ¹¤¯Ä󰯤µ¤ì¤Æ¤ª¤ê¡¢ÆÃÄê¤ÎCNSÎΰè¤Ë¤ª¤±¤ëÊÑÀ¥Ë¥å¡¼¥í¥ó¡Ê¿À·ÐºÙ˦¡Ë¤È´ØÏ¢¤·¤Æ¸«½Ð¤µ¤ì¤Æ¤¤¤ë¤³¤È¤Ï¶Ã¤¯¤Ù¤¤³¤È¤Ç¤Ï¤Ê¤¤¡£¥¢¥ë¥ß¥Ë¥¦¥àÃíÆþưʪ¤Ï¡¢Èé¼Á¤ª¤è¤Ó³¤ÇÏ¡ÊÃ𣲡ˤˤª¤±¤ë¥³¥ê¥óºîưÀËöü¤Î½ÅÅÙ¤ÎÁ°¿ÊÀÊÑÀ¤ò¼¨¤¹¡£
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The other is the Guamanian variant of ALS first described after World War 2 termed amyotropic lateral sclerosis parkinsonism dementia complex (ALS–PDC).¡¡This spectrum of disorders, once present with an incidence levels hundreds of times higher than in the continental United States [ 5 ] (see Kurland, 1988, for review), expressed in one of two ways ¤â¤¦°ì¤Ä¤Ï¡¢ÂèÆó¼¡À¤³¦ÂçÀï¸å¤ËºÇ½é¤ËµºÜ¤µ¤ì¤¿ALS¡Ê¸ÅŵŪ¶Ú°à½ÌÀ¦º÷¹Å²½¾É¡Ë¤Î¥°¥¢¥Þ¥Ë¥¢ÊѰÛÂΤǤ¢¤ê¡¢¶Ú°à½ÌÀ¦º÷¹Å²½¾É¤Î¥Ñ¡¼¥¥ó¥½¥Ë¥º¥àÃÔÊòÊ£¹çÂΡÊALS-PDC¡Ë¤È¸Æ¤Ð¤ì¤Æ¤¤¤ë¡£
¡Ê¥°¥¢¥àÅçÇÉʼʼ»Î¤Î¾ì¹ç¡Ë¤«¤Ä¤ÆÊƹñÂçΦ¤è¤ê¤â¿ôÉ´Çܤâ¹â¤¤È¯À¸Î¨¤òÄ褷¤Æ¤¤¤¿¤³¤Î¼ï¤Î¾ã³²¤Ï¡¢2¤Ä¤ÎÊýË¡¤Î1¤Ä¤Çɽ¸½¤µ¤ì¤¿¡ÊKurland¡¢1988¤ò»²¾È¡Ë Studies into potential etiologies focused on environmental factors with most attention eventually directed at the consumption of toxin-containing seeds of the local variety of cycad palm and the presence of high aluminum in the soil on southern Guam .
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The second was a form of parkinsonism associated with an Alzheimer's disease-like dementia (PDC).¡¡About 10% of the victims developed both disorders, with the ALS phenotype typically appearing first.
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Various studies have found no adverse or long-term health effects due to aluminum adjuvants [ 11 – 13 ] and the Food and Drug Administration (FDA) has continued its longstanding approval for the use of aluminum in this fashion.
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It also seems that there have been no rigorous animal studies of potential aluminum adjuvant toxicity.
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Summary of human ALS and GWI symptoms compared with symptoms observed in aluminum-treated mice and rats.¡¡This table also outlines the similarities between human ALS and Gulf War illness.
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However, the aluminum injected group showed a highly significant increase in the expression of GFAP positive astrocytes (70%) are the control group (listed as 100% for all graphs; Fig. 1C ) in the cervical segment of spinal cord. These GFAP results mirrored the outcomes previously reported in lumbar cord.
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AlCl3 significantly impaired learning and memory and increased brain AChE, brain total protein, TBARS, and nitrate/nitrite and decreased brain GSH or SOD.¡¡The likelihood exists that a synergistic effect between adjuvants and other variables such as stress, multiple vaccinations, and exposure to other toxins likely occurs.
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Evidence that mercury from silver dental fillings may be an etiological factor in smoking
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Author information
Physiology Department, Colorado State University, Fort Collins 80523.
Erratum in¡¡Toxicol Lett 1993 Sep;69(3):305
The smoking habits of 119 subjects without silver/mercury dental fillings were compared to 115 subjects with amalgams.¡¡The amalgam group had 2.5-times more smokers per group than the non-amalgam group, which was highly significant.¡¡Because mercury decreases dopamine, serotonin, norepinephrine, and acetylcholine in the brain, and nicotine has just the opposite effect on these neurotransmitters, this may help explain why persons with dental amalgams smoke more than persons without amalgams.
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